Ageing Lens

by SI
We have a highly elastic lens composed of crystallin proteins behind the iris of our eye. When functioning normally, changes in lens shape and thickness from action of the ciliary body, a circular muscle which also holds it in place, allow us to focus on near or distant objects.
The several types of crystallin proteins within maintain transparency, refractive power and flexibility, and filter higher bandwidths of light in the ultraviolet (UV) range. UV filtering prevents these biotoxic bandwidths from reaching the retina and damaging the delicate cells at the back of the eye.
Over time, ageing of lens epithelial cells at the front of the lens causes downstream effects, such as changes to the crystallin proteins. Studies show that this can be exacerbated by excess solar ionising radiation. The alteration of crystallin properties and function are behind the loss of elasticity and transparency in ageing lenses and the resultant development of cataracts.

Presbyopia

Presbyopia refers to the inability of an ageing eye to focus on near objects. It occurs due to normal age-related change to ocular muscles and lens elasticity. Presbyopia occurs regardless of LASIK or myopia at younger ages.

Glare & Other Visual Disturbances

Our crystallin lens is a structure that undergoes significant changes with age, eventually resulting in cataracts that are the world’s leading cause of (treatable, but so far not preventable) blindness.

Cataracts

Most cataracts are a normal part of ageing and cannot really be prevented, but they are treatable.

Risks from

Early Signs